Abstract
Twenty-four pigs were divided into two trials to determine if PSE musculature could be produced by aldosterone and prevented by an aldosterone-blocking agent. An intravenous injection of l.5 mg of aldosterone resulted in a significant (P<.01) reduction in Wisconsin pork quality score, and a trend toward decreased quality as measured by objective methods. The oral administration of 500 mg of Aldactazide caused a significant (P<.05) increase in Wisconsin pork quality score and a decrease in protein turbidity, both indicating superior muscle quality. Serum and muscle sodium and potassium contents were not affected significantly by any of the treatments imposed. Aldosterone was postulated to influence PSE conditions through its effect on muscle potassium, inactivating pyruvate kinase. However, muscle potassium values failed to substantiate this postulation.
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