Abstract
Inflammatory mediators are known to play a role in the progression of infectious models of inflammation, however, recent evidence suggests that the same mediators are responsible for the local and systemic inflammation in sterile animal models such as pancreatitis, ischemia-reperfusion or the injection of turpentine or zymosan. Using these noninfectious animal models, novel mediator-directed therapies can be critically evaluated and the subsequent data utilized to design possible clinical trials.
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