Abstract
Alzheimer’s disease is an age-related progressive neurodegenerative disorder leading to dementia. Numerous markers of inflammation, including major histocompatibility glycoproteins, cytokines and their receptors, classical pathway complement proteins and mRNAs, complement receptors and complement regulatory proteins, have been identified in brain samples taken from patients with Alzheimer’s disease. In contrast, such markers are not detected or are significantly reduced in the brains of nondemented elderly control patients.
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