Infective Endocarditis Caused by Pseudomonas stutzeri: A Case Report and Literature Review

Marwan J Alwazzeh,Yasser El-Ghoneimy,Feras A Alkuwaiti,Sarah Alwarthan,Moammer Alqasim

doi:10.3390/idr12030020

Abstract

Pseudomonas bacteria are widespread pathogens that account for considerable infections with significant morbidity and mortality, especially in hospitalized patients. The Pseudomonas genus contains a large number of species; however, the majority of infections are caused by Pseudomonas aeruginosa, infections by other Pseudomonas species are less reported. Pseudomonas stutzeri is a ubiquitous Gram-negative bacterium that has been reported as a causative agent of some infections, particularly in immunocompromised patients but has rarely been reported as a cause of infective endocarditis. Here, we report a case of a 55-year-old female with no significant medical history who presented with exertional dyspnea, productive cough, and fever. She was diagnosed as a case of acute anterior ST myocardial infarction, underwent double valve replacement surgery, and was found to have infective endocarditis caused by Pseudomonas stutzeri.

Highlights

A 55-year-old Saudi female, medically free, presented to the emergency room complaining of exertional dyspnea, New York Heart Association (NYHA) Class 2 accompanied by productive cough with whitish sputum
P. stutzeri, which was first described by Burri and Stutzer [1], is widely distributed in the environment
It is usually considered as colonization or contamination, but it has been reported as a causative agent of infections with different presentations

Summary

Introduction

A 55-year-old Saudi female, medically free, presented to the emergency room complaining of exertional dyspnea, New York Heart Association (NYHA) Class 2 accompanied by productive cough with whitish sputum. Echocardiography was done three hours post coronary angiography and showed the mitral valve with degeneration, moderately sized vegetation attached to the ventricular surface of the anterior leaflet (6 mm × 7 mm), and severe mitral regurgitation (Figure 1). The aortic valve showed moderate leaflet thickening, mild calcified trileaflet aortic valve, large-sized vegetation attached to the left coronary cusp (16 mm × 7 mm).

Results

Conclusion