Abstract
Unilateral lesions were placed in the entorhinal cortex of rats and the hippocampal formation examined histochemically for the localization of acetylcholinesterase. On the lesioned side, an intense band of acetylcholinesterase developed in the molecular layer of the dentate gyrus exactly where the entorhinal fibers normally terminate. This layer develops over a period of 30–40 days and is eliminated by septal lesions, which cause the disappearance of the normal bands of acetylcholinesterase. The suggestion is made that the sprouting of acetylcholinesterase containing septal terminals occurs under these conditions.
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