Abstract
Various types of neuronal damage have been reported in AIDS dementia complex (ADC). Human immunodeficiency virus (HIV) infected microglial/macrophage, not neuron directly in the brain. However, the damage of the /INS;neuron existed after the infection, in which the regulatory protein Tat of HIV-1 could be the critical factor. It has been recently reported that HIV-1 Tat transactivation requires menin, which/INS; hints that menin may be involved in the pathogenesis of ADC. However, the mechanism has not been elucidated completely.
Sign-in/Register to access full text options 
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a callDisclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.
