Abstract
Objectives: Patients with rheumatoid arthritis (RA) are characterized by a marked increase in cardiovascular risk, atributed to the systemic pro-inflammatory state. Whereas remission of RA can be achieved in the majority of patients, some require continuous biological therapy, which is potentially mediated by a chronic enhanced cellular drive. It is unclear whether remission in the joint inflammation coincides with attenuation of the inflammatory activity in the arterial wall. We hypothesized that RA patients requiring persistent biological therapy are characterized by a higher inflammatory state.
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