Abstract
Objective: Both PCOS and HT are syndromes defined by ovarian hyperandrogenism and chronic anovulation. In PCOS, increased LH secretion together with increased theca cell responsiveness to LH is responsible for excess androgen production. In contrast, serum LH levels in HT are normal and circulating androgen levels are generaly higher than those found in PCOS. These observations suggest that theca cell sensitivity to LH in HT is greater than that of PCOS. To begin to address this issue, we compared 17P responses to recombinant human LH (r-hLH) in HT and PCOS.
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