Abstract
Acute kidney injury (AKI) occurs frequently in the course of COVID-19 in the general population, its incidence ranging from 7% in hospitalized patients to above 20% in patients admitted to ICU. The definitive pathogenesis is still unclear. However, because in most cases the pattern of injury is acute tubular necrosis and endothelial injury, it is likely that most cases of AKI are the result of hemodynamic instability, cytokine-related injury, and dysfunction of the coagulation cascade.
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