Highlights

  • Streptococcus agalactiae (Group B streptococcus, Group B Streptococcus (GBS)) remains a leading cause of invasive infection in neonates [1]

  • Invasive GBS infection was diagnosed in 625 children (Incidence: 0.62 per 1000 livebirths; 95% confidence interval (CI): 0.57-0.67)

  • Information on GBS disease was retrieved from the Norwegian Surveillance System for Communicable Diseases (MSIS) and combined with information from The Medical Birth Registry of Norway (MBRN) regarding maternal health before and during pregnancy, the course of the delivery and newborn health, as well as with neurodevelopmental information retrieved from The Cerebral Palsy Registry of Norway (CPRN)

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Summary

Introduction

Streptococcus agalactiae (Group B streptococcus, GBS) remains a leading cause of invasive infection in neonates [1]. Despite improvements in maternal and perinatal care, GBS infection is still associated with substantial mortality and morbidity [2, 3]. Invasive infection presenting within the first seven days of life is classified as early onset disease (i.e. EOD), while infection presenting between seven and 89 days of life is classified as late onset (i.e. LOD). One to two percent of these children will develop invasive GBS infection [4]. Streptococcus (GBS) is the worldwide leading cause of invasive neonatal infection. We hypothesized that invasive GBS infection was associated with increased risk of infant mortality and cerebral palsy (CP). Invasive GBS infection during infancy was categorized into early onset disease (EOD), late onset (LOD) and very late onset disease (VLOD).

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