Abstract
The functional interaction between the immune and neuroendocrine system is mediated by humoral mediators, cytokines, neurotransmitters, and hormones. Indeed, the immune response is accompanied by homeostatic changes in the neuroendocrine system. The neuroendocrine changes produced by T-cell independent antigens are very different from those produced by T-cell dependent antigens. Several lines of evidence indicate that prolactin (PRL) and thyrotrophin releasing hormone (TRH) play important roles in T-cell dependent immune responses. This review focuses on the immunoregulatory functions of PRL, which is required for the induction of T-cell dependent immune response.
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