Abstract
Heart transplantation (HTx) is limited due to the availability of donor hearts.1 Up to 20% of patients die awaiting HTx.1 HTx numbers could increase by 56% if hearts donated after circulatory death (DCD) were used for transplantation.2 Recent trials have shown that DCD heart transplantation is feasible.3 However, innate to the DCD process is ischemia, ventricular wall stretching, and high catecholamine release. Upon implantation, reperfusion injury causes additional damage to the DCD heart.
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