Abstract
THE metabolic derangements of diabetes mellitus have been controlled by insulin therapy, but the degenerative changes in this disease continue to take their toll. The changes in the renal and retinal capillaries and the sequelae of these lesions are characteristic of diabetes mellitus. Controversy exists about the causative factors in these degenerative changes.1 The anterior pituitary gland and adrenal cortex have been implicated because of the following observations: the reported worsening of diabetic retinopathy during periods of stress2 , 3; the development of capillary aneurysms in nondiabetic patients during periods of treatment with ACTH and the disappearance of lesions after cessation . . .
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