Abstract

Dysmenorrhoea (usr-e-tams) is a usual gynaecological condition comprising of painful muscle spasms coming with menstruation, which in the absence of any fundamental irregularity or pathology, is known as primary dysmenorrhoea. Primary dysmenorrhoea is the nearly usual cyclical pelvic pain affecting the satisfaction of life. The relative incidence of primary dysmenorrhoea was reported to be within 20% and 90% in many societies. Studies have shown that regular exercise decreases dysmenorrhoea in women, which may be due to the effects of hormonal changes on uterine epithelial tissues or an enhancement in endorphin levels. It appears that exercise has pain-killing events that act in a non-particular way. Research has demonstrated that women with dysmenorrhoea have high degrees of prostaglandins, and hormones acknowledged to cause hampering abdominal pain. Exercise is a non-pharmacological treatment with the aim of reducing side effects commonly reported in association with NSAIDs, such as indigestion, headaches and drowsiness. Thus, diminution of pain may be due to consequences of hormonal changes on uterine epithelial tissues or an increase in endorphin levels. However, exercise has analgesic events that behave in a non-specific way and may be substituted for analgesics. Exercise may play a pivotal role in combating difficulties due to dysmenorrhoea. This article reviews the effects of exercise on dysmenorrhoea.

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