Abstract
Low forced vital capacity (FVC), decreased carbon monoxide diffusing capacity, and hypoxemia have been linked to poor prognosis in pulmonary fibrosis. The restrictive physiology may result in an increased respiratory rate, lower carbon dioxide tension (PaCO2), and less functional transplanted lung volume. We hypothesize that a low recipient PaCO2 at the time of listing negatively impacts post-lung transplant survival in patients with pulmonary fibrosis.
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