Abstract
Human predisposition to neurological diseases such as schizophrenia, Alzheimer’s, Parkinson’s and other neuropathologies is associated with genetic and environmental factors. One of the promising directions in the area of molecular neurosciences is the study of the role of immunogenetic mechanisms in different types of pathological processes in brain. This review explores the role of complex histocompatibility genes in the pathogenesis of schizophrenia, evaluating changes in the immune repertoire of T- and B-cell receptors in neuroinflammation.
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