Abstract
Long-term outcome of kidney transplantation is limited by the development of allograft rejection due to activation and realization of Tand B-cell immune response as well as the effects of other types of immune reactions mediated by various mechanisms. This article is dedicated to nowadays point of view upon the different immune and morphological phenotypes of kidney transplant injury. Main focus is made on the pathways of microvascular inflammation in the case of donor-specific antibodies absence as the unique self-sustained phenotype of the kidney allograft injury.
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