Abstract
Introduction and objective: Postoperative hyponatraemia (HN) cause serious morbidity and mortality, of which the transurethral resection of the prostate (TURP) syndrome is a unique model. Clinical presentation is circulatory shock and multiple vital organ dysfunction/ failure (MVOD/F) or death. All severe cases were reported retrospectively and attributed to multiple toxic/ dilution hypotheses interchangeably with recognized clinical conditions. The overlooked VO/T causes dynamic HN nadirs and masks making it a complex clinical and biochemical jigsaw puzzle. The objective here is to resolve this puzzle.
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