Abstract
The presence of rare pathogenic SCN5A variants is associated with more severe phenotypes in Brugada syndrome patients, which may be at least partly explained by greater epicardial arrhythmogenic substrates in these patients. The presence of rare pathogenic SCN5A variants is associated with more severe phenotypes in Brugada syndrome patients, which may be at least partly explained by greater epicardial arrhythmogenic substrates in these patients.
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