Abstract

WASHINGTON, D.C. --Like a living room wall with holes that have been plastered and painted over, cells from people with a premature aging disease can look normal again when the product of the culprit gene is fixed, according to research presented here 6 December 2004 at the American Society for Cell Biology Annual Meeting. The results verify the idea that the faulty protein prevents the normal one from doing its job and provide clues to possible therapy.

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