Abstract
Correction: Unfortunately, several authors on this paper were omitted. Therefore, on 15th April 2019 the following authors were added to the paper: Mathew Ibrahim Amprayil, Rahul Taparia and Shailesh Mani Pokhrel. The editorial board apologises for this error.
 The common risk factors for Idiopathic intracranial hypertension are obesity, female gender, hypervitaminosis A, and steroid withdrawal. Even though Idiopathic intracranial hypertension is considered as a neuropsychiatric manifestation of Systemic lupus erythematosus, it is often missed by the physician as a cause of a headache in a patient with Systemic lupus erythematosus. We report a case of 21-year-old female who presented in our outpatient department with a history of a severe intractable progressive headache for a duration of four weeks and blurring of vision for five days who was later diagnosed as a case of idiopathic intracranial hypertension with Systemic lupus erythematosus. She recovered dramatically with the institution of steroid and acetazolamide therapy.
Highlights
The prevalence of Idiopathic intracranial hypertension (IIH) or pseudotumor cerebri in the Western world was 0.9/100000 person in females of 15-44 years age group.[1]
We report a case of 21-year-old female who presented with severe intractable headache and diagnosed as a case of Systemic lupus erythematosus with idiopathic intracranial hypertension
The photograph of the fundus taken by Optical Coherence Tomography (OCT) after two weeks of treatment with steroid and acetazolamide is shown in figure 3. (JAIM): 14th issue, Volume 07, Number 02; July - December 2018
Summary
The prevalence of Idiopathic intracranial hypertension (IIH) or pseudotumor cerebri in the Western world was 0.9/100000 person in females of 15-44 years age group.[1]. There was no history of the seizure, trauma to head and fever She had visited two nearby hospitals prior to presenting to our centre. She was prescribed Tablet Ibuprofen 400 mg per oral thrice a day and tablet tramadol 50 mg once a day but that didn’t relieve her headache. History did not reveal a chronic headache and autoimmune disease in the first-degree relative On examination, she was conscious, oriented to time, place and person. We discharged the patient on oral prednisolone 50 mg once a day, tablet acetazolamide 500 mg thrice a day and tablet folic acid 5 mg once a day after five days of hospital admission. The patients had a follow-up visit two weeks after hospital discharge and there was a significant improvement in her headache and vision. The photograph of the fundus taken by Optical Coherence Tomography (OCT) after two weeks of treatment with steroid and acetazolamide is shown in figure 3
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