Abstract
Identification of Myomaker in Yellowfin Seabream (Acanthopagrus latus) (Hottuyn, 1782) and its Transcriptional Regulation by Two MyoDs
Highlights
Lactose non-persistence (LNP) develops due to the downregulation of the lactase-phlorizin hydrolase (LPH) enzyme
Molecular identification and screening in LNP subjects provide the mutational variants of the LCT-gene in the region of Pakistan
Whereas in the present study, the reported age of onset for this enzymatic deficiency varies from 2-17 years (Cook, 1967; which occurs after weaning in different populations Lehtimaki et al, 2006), the highest prevalence in Pakistani (EFSA, 2010)
Summary
Actose non-persistence (LNP) (OMIM#223100) is a genetically determined, developmental phenomenon, the reports, Finnish children had an onset age from 0-20 years (Rasinpera et al, 2004; Vesa et al, 2000). In the past, caused by decreased lactase enzyme production It can LNP was diagnosed with help of tests like glucose-oxidase cause different kinds of intestinal and systemic symptoms: test, blood glucose test, lactose tolerance test, glucoseheadache, vomiting, nausea, flatulence, abdominal pain, galactose tolerance test, urinary galactose test, stool pH gut distension, diarrhea, constipation, loss of concentration, and breath hydrogen test. All of these tests did not provide muscle pain, allergies, heart arrhythmia, mouth ulcers, sore reliable results
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