Abstract
Objective: Endometriosis is one of the most common diseases in women at reproductive age. It is also one of the major causes of infertility. There is yet no conclusion on the etiology and pathogenesis of endometriosis. It is hypothesized in this study that specific genetic alterations and/or aberrant expression of specific genes in endometrial cells is the underlying molecular mechanism for successful implantation and growth of the disseminated endometrial cells to form endometriosis. This project focused on the differential gene expression in human endometriosis compared with the paired normal uterine endometrium.
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