Abstract
Excess adiposity related chronic disease is the new medical term for obesity reflecting adipose tissue dysfunction as the sinister culprit in metabolic disease pathophysiology and burden. Visceral fat volume and inflammation, drained by the portal circulation, has been shown to have a critical role in metabolic disease, yet is difficult to quantify or sample via non-invasive modalities given deep mesenteric location (Figure 1). We hypothesize that aspects of visceral fat can be reliably measured during routine endosonography (EUS) of the gastrohepatic ligament, by estimating the celiac artery mesenteric fat thickness (CAMEUS) and this can correlate with metabolic disease burden.
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