Abstract
The parasympathetic modulation of gastrointestinal (GI) functions is accomplished by the activation of either vagal nerve (from esophagus to mid-colon) or sacral nerve (distal colon and anorectum). However, recent studies reported the role of auricular vagal nerve stimulation (aVNS) in controlling motility of the distal colon and rectum, suggesting a crosstalk between vagal and sacral nerves. The aim of this study was testing the hypothesis that exogeneous VNS is capable of altering functions of the distal colon and rectum that are not innervated with the vagus nerve.
Published Version
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