Abstract
Despite temporal lobe epilepsy (TLE) being a focal syndrome, patients experience widespread brain deficits. It has been shown previously that intrinsic connectivity networks (ICNs) have abnormal connectivity in epilepsy patients. Additionally, we have previously shown abnormal connectivity in arousal structures in TLE. Arousal structures may modulate these ICNs. Here, we investigate if patients with TLE have perturbations in functional connectivity (non-directed and directed) between arousal structures and ICNs.
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