Abstract
Spinal cord stimulation (SCS) is a safe and effective treatment option for patients with chronic intractable pain. However, a portion of patients with SCS eventually develop loss of efficacy for several reasons including habituation, lead migration, and new pain emergence. Burst stimulation offers a unique mechanism of action that can potentially restore effectiveness where other stimulation therapies have failed. While tonic SCS modulates the lateral pain pathway, only passive recharge burst1 has been shown to influence both the lateral and medial pain pathway responsible for affective components of pain, such as catastrophizing and depression in addition to sensory pain.
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