Abstract
Background: Dental agenesis is a term that refers to the absence of one or more teeth, and hypodontia refers to a severe type of tooth agenesis involving less than six or more than one congenitally missing teeth, excluding the third molars. Purpose: This aimed to report the correction of overbite by using intrusion archwires. Case: A 20-year-old female patient had class I malocclusion and deep overbite, incisor retroclination, mild mandibular crowding and agenesis of 12, 13, 14, 15, and 24. Case management: The case was treated with non-extraction using 0.022 pre-adjusted technique to level and unravel using intrusion archwires to correct the deep overbite. Conclusion: The 17-month treatment resulted in a corrected overbite, good occlusion, and good facial aesthetics.
Highlights
Dental agenesis is one of the most common developmental disorders and is indicated with the absence of one or more than one tooth
The majority of case reports showed that dental agenesis is mostly caused by genetic and molecular factors
(1) The incidence of agenesis varies from 1.6 to 9.6%, except for the loss of third molars, which occurs in 20% of the population.[2,3] Evaluation study of permanent dental agenesis can be performed by clinical examination and radiographic evaluation of the oral cavity.[4]
Summary
Dental agenesis is one of the most common developmental disorders and is indicated with the absence of one or more than one tooth. In the case of malocclusion with lower second premolar agenesis without crowding, and the primary second molars are in good condition (no caries and root resorption), the primary second molar can be maintained in the arch. Maintaining these teeth can prevent space closure without tipping and periodontal problems. This paper aimed to report the management of agenesis by keeping the primary teeth to maintain the space, correct the overbite, and achieve good facial aesthetic using intrusion arch wires. The lower and upper face lengths were normal with intrusion of the central incisor and extrusion of the maxillary molar (Figure 8)
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