Abstract

Although several theories have been suggested for hypersensitive dentin, it appears that the hydrodynamic theory is most plausible. If a clinical diagnosis of dentin hypersensitivity can be made, occlusion of the patent tubules would result in a reduction of hypersensitivity by the rapid fluid shift within the tubules. The naturally occurring defense mechanisms would wall off the narrow tubules producing the fluid flow, thus reducing the pain associated with hypersensitivity. In Part 11 of this article the current treatment modalities for dentin hypersensitivity will be discussed.

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