Abstract
The first quantitative measurements of thickening of the horny layer of human skin in response to injurious stimuli were done by Miescher (1) who demonstrated such a reaction following ultraviolet light irradiations. There is good reason to believe that thickening of the horny layer is a general reaction following not only actinic but also mechanical, chemical, electrical, and possibly thermal stimulation. Banal examples of extreme thickening of the horny layer following chronic mechanical trauma are calluses and corns. Increasing resistance to repeated applications of caustic chemicals, probably based on horny layer thickening, has been reported (2). Hyperkeratosis of the sweat gland orifices after electrical stimulation was recently found by Shelley et al. (3).
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