Abstract
A critical sized defect is the smallest osseous wound that will not heal spontaneously with bony union over the lifetime of an animal. Practically, the defect should not heal spontaneously during the experimental period. Hyperbaric oxygen therapy (HBO) is known to increase neovascularization, capillary in-growth as well as alkaline phosphatase activity and calcium levels and is an effective adjunct to improve bone healing postoperatively even in non-irradiated tissues. This study is intended to evaluate whether the effects of HBO can alter the predicted size of the critical sized defect in the rabbit calvarial model.
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