Abstract
Celiac disease (CD) is an autoimmune disease that occurs in genetically predisposed individuals, characterized by a permanent intolerance to ingested wheat gluten or related proteins from rye and barley, and immunologically mediated inflammatory damage of the small intestinal mucosa [1]. Environmental factors such as gluten administration in childhood and breastfeeding play a synergetic role in the induction of disease. Serologic surveys have shown that CD is a common disease, possibly affecting ∼ 1% of the population [2], whereas overt CD is rare, suggesting a high proportion of subclinical disease. Here we report a case of CD who demonstrated persistently elevated levels of serum amylase and lipase, reactive plasmacytosis in the bone marrow, and hypergammaglobulinemia characterized by increased levels of serum immunoglobulin (Ig) A and IgG. Immunoprecipitation assay showed that amylase was bound to polyclonal
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