Abstract
The frequency and distribution of male recombination events induced in dysgenic hybrids of Drosophila melanogaster has been investigated. Two wild-type inbred lines extracted from the “Texas” population were shown to differ substantially in the location of chromosome breakage points, thereby providing evidence that populations display heterogeneity of integration sites for the transposable elements responsible for dysgenic interactions. An attempt has been made to relate these data to current views on the molecular basis of hybrid dysgenesis. Evidence is provided which suggests that male recombination, SF female sterility and GD female sterility are induced by the same mobile genetic elements, indicating an identiy of the P-M and I-R interaction systems. The basis of the reported independence of these systems is discussed.
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