Abstract
Beal et al (1) have postulated that abnormal metabolism of tryptophan or quinolinic acid underlies the mechanism which causes brain damage in Huntington's disease. A case history is reported where a low tryptophan diet was associated with an unexpectedly good outcome. It is possible that a low tryptophan diet may alter the course of Huntington's disease, either by postponing onset or diminishing the severity of symptoms.
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