Abstract
The human papilloma virus (HPV) is responsible for different pathological manifestations in humans. This agent gives rise to lesions of different types and in different areas of the organism, including the oral cavity. The aim of this study is to show which are the main diseases for which HPV is responsible and to bring to light some of the interceptive and therapeutic strategies. The analysis was conducted by consulting the major scientific databases with the aim of obtaining information on the characteristics of oral HPV and its management; furthermore, the literature was supported by some clinical cases proposed by the authors. The role of dentistry is essential in the early diagnosis of this type of pathologies and above all in knowing how to direct patients towards a path that can lead to patient management, especially in the event that these lesions have a malignant potential. Enhancing the knowledge and role of dentistry can lead to early diagnosis of this type of injury, intercepting a pathology that could have multiorgan implications.
Highlights
IntroductionPapilloma viruses (from the Latin “papilla” meaning pustule and from the Greek suffix “oma”) are classified in the Papillomaviridae family, officially recognized by the International Committee on Taxonomy of Viruses (ICTV)
Papilloma viruses are classified in the Papillomaviridae family, officially recognized by the International Committee on Taxonomy of Viruses (ICTV)
The human papilloma virus (HPV) is a small-sized DNA virus without envelope, which is resistant to heat, acids, and ether
Summary
Papilloma viruses (from the Latin “papilla” meaning pustule and from the Greek suffix “oma”) are classified in the Papillomaviridae family, officially recognized by the International Committee on Taxonomy of Viruses (ICTV). The human papilloma virus (HPV) is a small-sized DNA virus (diameter 50-55 nm) without envelope, which is resistant to heat, acids, and ether. Papilloma viruses have a high tropism for stratified squamous epithelial cells and replicate only in differentiating epithelial cells of the skin and mucous [2]. The infection begins with the entry, following small wounds or superficial abrasions of the host’s mucosa, of a viral particle into a cell of the basal epithelial layer called keratinocyte characterized by a marked proliferative activity. Once the virus has penetrated into the keratinocyte, it could remain in a latent state or undergo active replication. This virus is responsible for causing lesions of the affected tissues [4,5,6]
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