Abstract
Aim: The impact of ApoA-I, main protein of HDL, has been long examined. However, the impact of ApoA-I on atheroprotective action of dysfunctional HDL in diabetes is still limited. In this regard, we analyzed its consequences in important properties of HDL such as in vivo macrophage-specific reverse cholesterol transport (m-RCT) and ex vivo anti-oxidant protection in db/db mice
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