Abstract
BackgroundEvolution of endoscopic surgery provides equivalent results to open surgery with advantages of minimal invasive surgery. The literature on technique Uniportal Full endoscopic contralateral approach is scarce.MethodsThe endoscopic contralateral approach technique applies for patients presenting with double crush syndrome with foraminal and extraforminal stenosis. The key steps focus on contralateral ventral overriding superior articular process decompression, foraminal and extraforaminal discectomy, and lateral vertebral syndesmophyte decompression leading to enlargement of the contralateral foramen and extraforamen size.ConclusionThe Uniportal Full endoscopic contralateral approach is a good alternative to open surgery or minimally invasive microscopic surgery through direct endoscopic visualization of the entire route of exiting nerve with no neural retraction allowing both lateral recess and foraminal and extraforaminal decompression all in one approach.
Highlights
Evolution of endoscopic surgery provides equivalent results to open surgery with advantages of minimal invasive surgery
With the sublaminar corridor, drilling focuses on the under surface of the spinolaminar junction. It is suitable for patients with minimal central and lateral recess stenosis, and it conserves most parts of the interspinous ligament and spinous
The systematic drilling technique is used with a 3.5-mm coarse diamond endoscopic burr on the base of the spinous process and ventral to the interspinous ligament at the spinolaminar junction to create a sublaminar space above the ligamentum flavum over the top of neural elements with the open bevel pointing upwards (Figs. 4a, b and 5a)
Summary
The endoscopic contralateral approach technique applies for patients presenting with double crush syndrome with foraminal and extraforminal stenosis. The key steps focus on contralateral ventral overriding superior articular process decompression, foraminal and extraforaminal discectomy, and lateral vertebral syndesmophyte decompression leading to enlargement of the contralateral foramen and extraforamen size
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