Abstract
Vitamin B plays an important role in the methylation of DNA to help regulate gene expression, especially in children experiencing rapid cell growth phases, such as pregnant women or young children. In addition, folate deficiency in the periconceptional period can contribute to neural tube defects, while vitamin B12 deficits have been associated with acute megaloblastic anemia. This research examines the relationship between maternal plasma vitamin B levels with their children and the long-term impacts of deficits in vitamin B to the cognitive abilities of pattern reasoning, verbal fluency, and memory retrieval as a few examples of how their cognitive and psychological developments are assessed. Further investigations should focus on the specific genes potentially muted due to vitamin B deficiency and examine how to prevent folate vitamin B12 deficiency in developing/third world countries.
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