Abstract
1. 1. Daily administration of cortisone acetate (25 μg/g body wt/day) or dl-thyroxine (1 μg/g body wt/day) for 3 days to 8-day-old suckling mice induces a premature increase of intestinal glucoamylase activity. 2. 2. The maximal response is observed in the proximal intestinal third after thyroxine treatment and in the middle third after cortisone treatment. 3. 3. In adult mice, glucoamylase activity is still influenced by thyroxine but no longer by cortisone. 4. 4. Sucrase and trehalase activities are unaffected by thyroxine in adult mice. 5. 5. This study shows that only one intestinal α-glucosidase activity, namely glucoamylase, is controlled during the entire postnatal life by thyroxine.
Sign-in/Register to access full text options 
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a callMore From: Comparative Biochemistry and Physiology -- Part B: Biochemistry and Molecular Biology
Disclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.
