Abstract
The activity of leucocyte δ-aminolævulinic-acid (A.L.A.) synthase was monitored throughout a prolonged attack of acute intermittent porphyria in a 29-year-old woman. Clinical severity was associated with a pronounced increase in activity of this enzyme and high urinary excretion of A.L.A. Hæmatin therapy resulted in clinical improvement associated with a reduction in the activity of A.L.A. synthase and reduction in urinary excretion of A.L.A.
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