Abstract
improved survival in HIV-infected patients [ 1-4] . This allegedly beneficial viral interference has led to suggestions that artificial GBV-C superinfection might be used as a new therapeutic strategy in HIV infection [5], However, other studies have failed to show this association [6, 7], and the discordant findings have been the subject of much debate over the past few years. It has also been found that GBV-C viremia has a tendency to disappear over time in HIV-infected subjects who are followed up longitudinally. This phenomenon, in turn, is associated with accelerated disease progression [6-8]. Thus, the time point at which GBV-C viremia status is defined is important. In parallel, in vitro studies have lent support to the existence of the protective effects of GBV-C infection for HIVinfected individuals on several levels. A
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