Abstract
Aim: Soluble endoglin (sEng) is generated by the cleavage of the extracellular domain from membrane-bound endoglin in endothelial dysfunction-related pathologies, such as atherosclerosis. In addition, sEng was proposed to affect vascular endothelium function. With respect to hypercholesterolemia as a risk factor of endothelial dysfunction, we hypothesized that combination of high sEng levels and hypercholesterolemia will results in the development/aggravation of endothelial dysfunction.
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