Abstract
HERPES zoster, or zona, was well known to the ancients. It was not until the nineteenth century that herpes zoster was recognized as the cutaneous phase of a disease involving the sensory portion of the spinal nerves. Von Baerensprung in 1861 first demonstrated at autopsy inflammatory lesions in the posterior root ganglia. In 1900 the work of Head and Campbell 1 led to our current hypothesis of herpes zoster. The disease is now generally regarded as an acute specific infection caused by a neurotropic virus with a predilection for the posterior spinal ganglia or the extramedullary ganglia of the cranial nerves, conferring an immunity and running a definite clinical course accompanied with an erythematous vesicular eruption in the cutaneous or mucosal distribution of the sensory nerves. Although many observers in the past century reported cases of herpes zoster of a cranial nerve, chiefly of Gasserian origin, but including herpes oticus
Published Version
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