Abstract
The scale of the problem of hepatotoxicity of botanicals is reviewed. This problem was found to be very small, largely limited to idiosyncratic reactions (as opposed to intrinsic toxicity of herbal compounds for the liver), and hampered by the availability of high-quality evidence against intrinsic hepatotoxicity. Claims that herbal hepatotoxicity is common but hidden could not be substantiated. Various herbs sometimes considered hepatotoxic are reviewed in depth, including Piper methysticum (kava); Larrea tridentata (creosote bush); unsaturated pyrrolizidine alkaloid–containing herbs, such as Symphytum officinale (comfrey) and Tussilago farfara (coltsfoot); Cimicifuga racemosa (black cohosh); and Camellia sinensis (green tea) and its catechins.
Sign-in/Register to access full text options 
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a callDisclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.
