Abstract
Acute myocardial infarction is a potential life threatening disease spread all over the world. The continuing progress of medical and interventional therapies requires a comprehensive understanding of the underlying pathophysiology. Moreover, the potential development of heart failure or/and arrhythmias in either acute or chronic setting, demand a deep knowledge of their molecular mechanisms in order to provide adequate treatments. This review aims to summarize the current data regarding the etiopathogenesis of acute myocardial infarction and the heart rhythm disorders associated with this clinical condition.
Highlights
ST-segment elevation acute myocardial infarction (STEMI), the most severe form of ischemic heart disease, is one of the leading causes of mortality in the world
Recent data have brought to the spotlight the role of inflammation in the development and further evolution of this severe pathology[3]
This review synthesizes the current knowledge in what concerns the pathophysiology of acute myocardial infarction (AMI) and the subsequent heart rhythm disorders, as well as possible future perspectives
Summary
ST-segment elevation acute myocardial infarction (STEMI), the most severe form of ischemic heart disease, is one of the leading causes of mortality in the world. This patient population usually dies from either heart rhythm disorders or heart failure[1,2]. The exact underlying pathophysiology of the acute myocardial infarction (AMI) has not been fully understood yet. Recent data have brought to the spotlight the role of inflammation in the development and further evolution of this severe pathology[3]. This review synthesizes the current knowledge in what concerns the pathophysiology of AMI and the subsequent heart rhythm disorders, as well as possible future perspectives
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