Abstract
Abstract Aim: The aim of the present study was to verify the agreement between the ventilatory method (VT) and the alternative method of heart rate deflection point (HRDP) in determining the anaerobic threshold (AT) during incremental treadmill test in dyslipidaemic patients. Methods: Twenty-seven dyslipidaemic patients (61.50 ± 10.46 years) performed an incremental treadmill test, in which the AT was determined using both methods. Bland-Altman statistics was adopted in order to verify the agreement between the methods. Results: Agreement in AT determination between the VT and HRDP methods was observed (p < 0.05) for heart rate (138.00 ± 23.80 and 136.26 ± 22.18 bpm, respectively), oxygen uptake (31.00 ± 10.33 and 31.00 ± 11.17 ml.kg−1.min−1), and treadmill velocity (7.67 ± 1.71 km.h-1and 8.00 ± 1.75 km.h-1). Conclusion: Our results suggest that the HRDP method can be adopted for the determination of the AT in dyslipidaemic patients, showing agreement with the VT method.
Highlights
Dyslipidaemias can be defined as heterogeneous disorders in lipid metabolism, which result in alterations in blood lipoprotein and lipid concentrations[1]
The results of the present study demonstrated that there is an agreement between HR, VO2 and velocity corresponding to the anaerobic threshold (AT) determined by the heart rate deflection point (HRDP) and ventilatory method (VT) methods
The Bland-Altman analysis suggests an acceptable concordance between the HRDP and VT methods for determination of the AT during an incremental treadmill test performed by elderly dyslipidaemic patients
Summary
Dyslipidaemias can be defined as heterogeneous disorders in lipid metabolism, which result in alterations in blood lipoprotein (low and high-density lipoprotein, LDL, and HDL respectively) and lipid concentrations (total cholesterol and triglycerides, TC and TG, respectively)[1]. This disorder is considered the main risk factor for the development of atherosclerotic cardiovascular disease, increasing the risk of cardiovascular events, such as myocardial infarction, stroke, and peripheral vascular disease[2]. Mikus et al.[10] have shown that statins users have lower chances to increase their cardiorespiratory fitness and skeletal muscle citrate synthase activity after a period of physical training
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