Abstract
Headache occurs in only about 13% of patients within the cohort of presenting COVID-19 symptoms. The hypothesis that such a painful symptomatic picture could be considered a prognostic factor for COVID-19 positive evolution or its trend of severity, or the co-generation of hyposmia/anosmia and/or hypogeusia/ageusia, needs robust epidemiological data, punctual pathophysiological demonstrations, and a detailed comparative analysis on drug–drug interactions (DDIs).
Highlights
To date, scientific literature is regrettably and exceedingly generous towards the term “COVID-19”: 71,664 scientific articles published [1], 49,578,590 confirmed cases, and 1,245,717 confirmed deaths for COVID-19 in less than 1 year [2]
As the number of new registered cases continues to rise on a daily basis, 441,696 on the 9th of November 2020, so too is there an increase in questions for the scientific community while certainties struggle to emanate from this immensity of scientific data
It is clear that all systems have been crushed by this cytokine release syndrome (CRS) and that the list of epidemiological and clinical characteristics, including headache, does not appear to be subject to further updates
Summary
Scientific literature is regrettably and exceedingly generous towards the term “COVID-19”: 71,664 scientific articles published [1], 49,578,590 confirmed cases, and 1,245,717 confirmed deaths for COVID-19 in less than 1 year [2]. Headache as a symptom of COVID-19 is present in only 12.9% of patients afferent to COVID-19 emergency medicine [3].
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