Abstract
It was shown that glutamate (10 μ M to 1 μM) suppresses in a dose-dependent manner the non-quantum release of acetylcholine from rat motor nerve endings; the release intensity was estimated by the H effect. The action of glutamate was completely eliminated by the blockade of guanylyl cyclase by 1 μM ODQ. An increase in the intracellular cGMP concentration by 1 μ M dibutyryl-cGMP reduced the H effect in a similar manner as glutamate did.
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