Abstract
We read with interest the recent genome-wide association study by Li and colleagues1 and the identification of single nucleotide polymorphisms (SNPs) in the glypican 5 (GPC5) gene associated with susceptibility to lung cancer in non-smokers. These variants were elegantly linked to the expression of GPC5, and this gene was also shown to be expressed at lower levels in lung cancer than in healthy lung tissue. The authors point out that different SNPs in GPC5 have also been implicated in susceptibility to multiple sclerosis (MS).
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