Abstract
In the adipose tissue like in the liver, glycogen molecules are synthesized in a molecular order such that some molecules are completed before others start to grow. In the adipose tissue, the degradation of glycogen occurs at random, whereas, in the liver, molecules that were synthesized last are degraded first and vice versa. This difference in the catabolism of glycogen between the two tissues may be related to the fact that glycogen is in the form of monomeric beta particles in the adipose tissue and of polymeric alpha particles in the liver.
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